If we had a proper patrolling police force of the old kind, many of these incidents would never happen. Such a force would apply the boring laws on drug possession which our armed and scowling gendarmes, and their soppy, pseudo-intellectual chief officers, think are beneath them. Through their intimate knowledge of their beats and their frequent contact with the law-abiding, they would be aware of the strange behaviour of such people long before it became a danger.E.G. being a real peace officer like advocated by the Detroit threat management center, and other private police forces. As always, I advocate for privatization, as they have little to no incentive to become this vicious occupying army like the cops are.
For an unarmed, modest, old-fashioned police force, which walks quietly among us, has millions of willing eyes and ears, in the shape of a friendly and supportive public.
But an armed state militia, dressed for combat with its face set in a rigid frown and its hands ever reaching for gun, club or handcuffs, such as we now have, is a stranger to the people. And as well as making us look like a foreign despotism, it will fail in its task.
Our results suggest that fishing at the current scale is enabled by large government subsidies, without which as much as 54% of the present high-seas fishing grounds would be unprofitable at current fishing rates. The patterns of fishing profitability vary widely between countries, types of fishing, and distance to port. Deep-sea bottom trawling often produces net economic benefits only thanks to subsidies, and much fishing by the worldβs largest fishing fleets would largely be unprofitable without subsidies and low labor costs. These results support recent calls for subsidy and fishery management reforms on the high seas.Lefties shoot themselves in the foot as usual. Destroy the environment to "help workers". Idiots.
βWe found that cutting the genome with CRISPR-Cas9 induced the activation of β¦ p53,β said Emma Haapaniemi, the lead author of the Karolinska study. That βmakes editing much more difficult.βYet more trouble for the approach. I think the knowledge about the new type of DNA needs to be expanded before we truly understand this.
The flip side of p53 repairing CRISPR edits, or killing cells that accept the edits, is that cells that survive with the edits do so precisely because they have a dysfunctional p53 and therefore lack this fix-it-or-kill-it mechanism.
The reason why that could be a problem is that p53 dysfunction can cause cancer. And not just occasionally. P53 mutations are responsible for nearly half of ovarian cancers; 43 percent of colorectal cancers; 38 percent of lung cancers; nearly one-third of pancreatic, stomach, and liver cancers; and one-quarter of breast cancers, among others.